<P>About 40% of central nervous system synapses use glutamate as the neurotransmitter. Over-stimulation of glutamate receptors produces neuronal injury or death by excitotoxicity, which is closely associated with neurochemical and neuropathological changes involved in acute neural trauma (stroke, sp
Neurochemical Aspects of Hypothalamic Function
β Scribed by L. Martini (Eds.)
- Publisher
- Elsevier Science
- Year
- 1970
- Tongue
- English
- Leaves
- 162
- Category
- Library
No coin nor oath required. For personal study only.
β¦ Table of Contents
Content:
Inside Front Cover, Page ii
Front Matter, Page iii
Copyright, Page iv
CONTRIBUTORS, Page vii
PREFACE, Pages ix-x, Luciano Martini, Joseph Meites
MODIFICATION OF SYNTHESIS AND RELEASE OF HYPOTHALAMIC RELEASING FACTORS INDUCED BY EXOGENOUS STIMULI, Pages 1-43, Joseph Meites
MODIFICATION OF SYNTHESIS AND RELEASE OF HYPOTHALAMIC RELEASING FACTORS INDUCED BY CHANGES OF THE ENDOCRINE βMILIEUβ, Pages 45-60, Marcella Motta
PARTICIPATION OF CENTRAL MONOAMINERGIC NEURONS IN THE REGULATION OF ANTERIOR PITUITARY SECRETION, Pages 61-83, Kjell Fuxe, Tomas HΓΆkfelt, GΓΆsta Jonsson
ROLE OF BRAIN CAT EC HOL AMINES IN THE CONTROL OF ANTERIOR PITUITARY FUNCTIONS, Pages 85-100, Claude Kordon, Jacques Glowinski
EFFECTS OF ENDOCRINE MANIPULATIONS ON THE METABOLISM OF HYPOTHALAMIC MONOAMINES, Pages 101-133, Walter Lichtensteiger
CONTROL OF THE SYNTHESIS OF MELATONIN AND OTHER METHOXYINDOLES IN THE MAMMALIAN PINEAL ORGAN, Pages 135-140, Richard J. Wurtman
ROLE OF INDOLEAMINES IN THE CONTROL OF THE SECRETION OF PITUITARY GONADOTROPINS, Pages 141-159, Franco Fraschini
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Leading researchers offer cutting-edge information on glutamate metabolism in the brain, examining the role of glutamate transporters and the involvement of glutamate receptors in the pathogenesis of acute neural trauma and neurodegenerative diseases. In addition, the authors discuss the treatment o
<p><P>About 40% of central nervous system synapses use glutamate as the neurotransmitter. Over-stimulation of glutamate receptors produces neuronal injury or death by excitotoxicity, which is closely associated with neurochemical and neuropathological changes involved in acute neural trauma (stroke,