## Abstract Zinc is an endogenous N‐methyl‐D‐aspartate (NMDA) receptor blocker. It is possible that zinc‐mediated modification of hippocampal CA1 long‐term potentiation (LTP) is linked to the expression of NMDA receptor subunits, which varies with postnatal development. In the present study, the ef
N-methyl-d-aspartate receptor-dependent long-term potentiation in CA1 region affects synaptic expression of glutamate receptor subunits and associated proteins in the whole hippocampus
✍ Scribed by W.X. Zhong; Z.F. Dong; M. Tian; J. Cao; L. Xu; J.H. Luo
- Book ID
- 116787818
- Publisher
- Elsevier Science
- Year
- 2006
- Tongue
- English
- Weight
- 918 KB
- Volume
- 141
- Category
- Article
- ISSN
- 0306-4522
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Among the different steroids found in the brain, pregnenolone sulfate (3β‐hydroxy‐5‐pregnen‐20‐one‐3‐sulfate; PREGS) is known to enhance hippocampal‐associated memory. The present study employs rat hippocampal slices to investigate the ability of PREGS to modulate long‐term potentiation
Evoked potentials recorded from slices of pigeon hippocampus were reversibly attenuated by 2 mM kynurenic acid. High frequency stimulation (3 x 200 Hz for 1 sec, with 1 sec intervals) evoked a persistent increase in the evoked potential, lasting in a nondecremental form for at least 2 hr. This incre