𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Myocardial tolerance to ischemia–reperfusion injury, training intensity and cessation

✍ Scribed by Fabio Esposito; Raffaella Ronchi; Giuseppina Milano; Vittoria Margonato; Simona Di Tullio; Marina Marini; Arsenio Veicsteinas; Michele Samaja

Publisher
Springer
Year
2010
Tongue
English
Weight
366 KB
Volume
111
Category
Article
ISSN
1439-6319

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Reduced L-arginine transport contributes
Reduced L-arginine transport contributes to the pathogenesis of myocardial ischemia-reperfusion injury
✍ Kylie M. Venardos; Amanda J. Zatta; Tanneale Marshall; Rebecca Ritchie; David M. 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 232 KB

## Abstract Myocardial injury due to ischemia‐reperfusion (I‐R) damage remains a major clinical challenge. Its pathogenesis is complex including endothelial dysfunction and heightened oxidative stress although the key driving mechanism remains uncertain. In this study we tested the hypothesis that

Induction of heat-shock protein 72 in ra
Induction of heat-shock protein 72 in rat skeletal muscle does not increase tolerance to ischemia-reperfusion injury
✍ Sean Lille; Ching-Yuan Su; Thomas Schoeller; Hans Suchy; Sharon Lyons; Robert C. 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 536 KB

Ischemia-reperfusion injury is implicated in the failure of free flap and replant surgeries and is associated with the pathogenesis of a wide variety of clinical diseases including stroke, myocardial infarction, spinal injury, and compartment syndromes. We used a skeletal muscle flap model to test i