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Mutations in HNF-1α that result in maturity onset diabetes of the young retain wild-type activation of target genes but exhibit a selective defect in cooperative interactions with endodermal transcription factors

✍ Scribed by Joyce K. Divine; Theodore C. Simon


Book ID
119427481
Publisher
Elsevier Science
Year
2003
Tongue
English
Weight
187 KB
Volume
124
Category
Article
ISSN
0016-5085

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