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Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid β-peptide (1–42), H2O2 and kainic acid: implications for Alzheimer's disease

✍ Scribed by Hafiz Mohmmad Abdul; Rukhsana Sultana; Jeffrey N. Keller; Daret K St. Clair; William R. Markesbery; D. Allan Butterfield


Book ID
111179376
Publisher
John Wiley and Sons
Year
2006
Tongue
English
Weight
552 KB
Volume
96
Category
Article
ISSN
0022-3042

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