✦ LIBER ✦

MRI of mouse models of neurological disorders

✍ Scribed by Stasia A. Anderson; Joseph A. Frank

Publisher: John Wiley and Sons
Year: 2007
Tongue: English
Weight: 437 KB
Volume: 20
Category: Article
ISSN: 0952-3480
DOI: 10.1002/nbm.1167

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Mouse models for congenital disorders of

Mouse models for congenital disorders of glycosylation

✍ Christian Thiel; Christian Körner 📂 Article 📅 2011 🏛 Springer 🌐 English ⚖ 615 KB

3He MRI in mouse models of asthma

✍ Bastiaan Driehuys; Julia Walker; Jim Pollaro; Gary P. Cofer; Nilesh Mistry; Davi 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 378 KB

## Abstract In the study of asthma, a vital role is played by mouse models, because knockout or transgenic methods can be used to alter disease pathways and identify therapeutic targets that affect lung function. Assessment of lung function in rodents by available methods is insensitive because the

Non-invasive MRI of mouse models of athe

Non-invasive MRI of mouse models of atherosclerosis

✍ David B. Weinreb; Juan Gilberto S. Aguinaldo; Jonathan E. Feig; Edward A. Fisher 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 539 KB

Neurological correlates of eating disord

Neurological correlates of eating disorders

✍ Uttom Chowdhury; Bryan Lask 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 English ⚖ 67 KB 👁 2 views

Behavioral profiles of mouse models for

Behavioral profiles of mouse models for autism spectrum disorders

✍ Elodie Ey; Claire S. Leblond; Thomas Bourgeron 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 525 KB

α-Synuclein abnormalities in mouse model

α-Synuclein abnormalities in mouse models of peroxisome biogenesis disorders

✍ Eugenia Yakunin; Ann Moser; Virginie Loeb; Ann Saada; Phyllis Faust; Denis I. Cr 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 330 KB

## Abstract α‐Synuclein (αS) is a presynaptic protein implicated in Parkinson's disease (PD). Growing evidence implicates mitochondrial dysfunction, oxidative stress, and αS–lipid interactions in the gradual accumulation of αS in pathogenic forms and its deposition in Lewy bodies, the pathological