## Abstract The presence of amyloid‐β (Aβ) plaques in the brain is a hallmark pathological feature of Alzheimer's disease (AD). Transgenic mice overexpressing mutant amyloid precursor protein (APP), or both mutant APP and presenilin‐1 (APP/PS1), develop Aβ plaques similar to those in AD patients, a
MR Microimaging of amyloid plaques in Alzheimer’s disease transgenic mice
✍ Scribed by Thomas M. Wengenack; Clifford R. Jack; Michael Garwood; Joseph F. Poduslo
- Publisher
- Springer
- Year
- 2008
- Tongue
- English
- Weight
- 442 KB
- Volume
- 35
- Category
- Article
- ISSN
- 0340-6997
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