𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Molecular genetic diagnosis of de novo and recurrent bladder cancer

✍ Scribed by Gabriel Steiner; Gudrun Reinschmidt; Stefan C. Müller

Publisher
John Wiley and Sons
Year
1999
Tongue
English
Weight
83 KB
Volume
20
Category
Article
ISSN
0173-0835

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Genetic studies and molecular markers of
Genetic studies and molecular markers of bladder cancer
✍ Carlos Cordon-Cardo; Joel Sheinfeld; Guido Dalbagni 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 88 KB 👁 1 views

Target genes implicated in cellular transformation and tumor progression have been divided into two categories: proto-oncogenes which, when activated, become dominant events characterized by the gain of function, and tumor suppressor genes which become recessive events characterized by the loss of f

Clinical impact of molecular genetic dia
Clinical impact of molecular genetic diagnosis, genetic counseling, and management of hereditary cancer : Part I: Studies of cancer in families
✍ Henry T. Lynch; Patrice Watson; Trudy G. Shaw; Jane F. Lynch; Anne E. Harty; Bar 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 164 KB 👁 2 views

Hereditary cancer represents approximately 5-10% of the total cancer burden and may account for 60,000 to 120,000 new cancer occurrences this year in the United States. New developments in molecular genetics and the cloning of cancer-prone genes have intensely fueled interest in dealing with heredit

Clinical impact of molecular genetic dia
Clinical impact of molecular genetic diagnosis, genetic counseling, and management of hereditary cancer : Part II: Hereditary nonpolyposis colorectal carcinoma as a model
✍ Henry T. Lynch; Patrice Watson; Trudy G. Shaw; Jane F. Lynch; Anne E. Harty; Bar 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 114 KB 👁 2 views

Hereditary nonpolyposis colorectal carcinoma (HNPCC) is the most common hereditary form of colorectal carcinoma (CRC) and may account for 5-10% of the total CRC burden. The discovery of DNA mismatch repair (MMR) genes, inclusive of hMSH2, hMLH1, hPMS2, and hMSH6, has enabled the identification of wh