✦ LIBER ✦

MLK3 is critical for breast cancer cell migration and promotes a malignant phenotype in mammary epithelial cells

✍ Scribed by Chen, J; Miller, E M; Gallo, K A

Book ID: 109972390
Publisher: Nature Publishing Group
Year: 2010
Tongue: English
Weight: 975 KB
Volume: 29
Category: Article
ISSN: 0950-9232
DOI: 10.1038/onc.2010.198

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Antimicrobial protein hCAP18/LL-37 is hi

Antimicrobial protein hCAP18/LL-37 is highly expressed in breast cancer and is a putative growth factor for epithelial cells

✍ Johan D. Heilborn; Margareta Frohm Nilsson; Clara I. Chamorro Jimenez; Bengt San 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 French ⚖ 537 KB

## Abstract Human cathelicidin antimicrobial protein hCAP18/LL‐37 is an effector molecule of the nonspecific innate immune system. hCAP18/LL‐37 is present in leukocytes and is expressed in skin and other epithelia, where it is upregulated in association with inflammation and injury. In addition, an

ADAMTSL3/punctin-2, a gene frequently mu

ADAMTSL3/punctin-2, a gene frequently mutated in colorectal tumors, is widely expressed in normal and malignant epithelial cells, vascular endothelial cells and other cell types, and its mRNA is reduced in colon cancer

✍ Bon-Hun Koo; Tiina Hurskainen; Katrina Mielke; Phyu Phyu Aung; Graham Casey; Hel 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 French ⚖ 473 KB

## Abstract ADAMTSL3/punctin‐2 is a secreted glycoprotein that resembles the ADAMTS proteases. Recently, identification of frequent __ADAMTSL3__ mutations in colorectal cancer suggested it might have a regulatory role in cellular homeostasis in colorectal epithelium or in pathways to colorectal mal

Influence of a Self-Assembling Peptide,

Influence of a Self-Assembling Peptide, RADA16, Compared with Collagen I and Matrigel on the Malignant Phenotype of Human Breast-Cancer Cells in 3D Cultures and in vivo

✍ Kun Mi; Guixia Wang; Zijia Liu; Zhihua Feng; Ben Huang; Xiaojun Zhao 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 508 KB

## Abstract Cancer‐cell phenotype is not only the result of malignant progression, but also dependent on the microenvironment surrounding them, including influences from the extracellular matrix and its structural properties. We have investigated the influence of the nanofiber matrix of the self‐as