๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Mitochondrial Involvement in Causing Cell Injury in Experimental Hepatic Iron Overload

โœ Scribed by A. MASINI; T. TRENTI; D. CECCARELLI; U. MUSCATELLO


Book ID
118724305
Publisher
John Wiley and Sons
Year
1986
Tongue
English
Weight
134 KB
Volume
488
Category
Article
ISSN
0890-6564

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Hepatic mitochondrial malondialdehyde me
โœ Robert S. Britton; Rosemary O'Neill; Bruce R. Bacon ๐Ÿ“‚ Article ๐Ÿ“… 1990 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 614 KB

Peroxidative decomposition of mitochondrial membrane phospholipids with subsequent mitochondrial dysfunction is a postulated mechanism of liver cell injury in parenchymal iron overload. Malondialdehyde is formed when polyunsaturated fatty acids of membrane phospholipids undergo peroxidative decompos

Hepatic mitochondrial oxidative metaboli
โœ Edward S. Feldman; Bruce R. Bacon ๐Ÿ“‚ Article ๐Ÿ“… 1989 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 713 KB

Both human porphyria cutanea tarda and experimental hexachlorobenzene-induced porphyria are associated with hepatic injury and are potentiated by excess hepatic iron. The mechanisms whereby cellular injury occurs and the synergistic role of iron overload are unknown. In the present experiments, we s