𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Methamphetamine-induced neurotoxicity and microglial activation are not mediated by fractalkine receptor signaling

✍ Scribed by David M. Thomas; Dina M. Francescutti-Verbeem; Donald M. Kuhn


Book ID
111180904
Publisher
John Wiley and Sons
Year
2008
Tongue
English
Weight
626 KB
Volume
106
Category
Article
ISSN
0022-3042

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Fractalkine modulates TNF-Ξ± secretion an
✍ Violetta Zujovic; JesΓΊs Benavides; Xavier VigΓ©; Christopher Carter; VΓ©ronique Ta πŸ“‚ Article πŸ“… 2000 πŸ› John Wiley and Sons 🌐 English βš– 362 KB

Among the chemokine family, fractalkine shows unusual properties: it exists as a membrane-bound and soluble protein, and both fractalkine and its receptor CX 3 CR1 are expressed predominantly in the central nervous system. In rat cell culture models, the chemokine fractalkine was expressed in neuron

P2Y12 receptor-mediated integrin-Ξ²1 acti
✍ Keiko Ohsawa; Yasuhiro Irino; Tomomi Sanagi; Yasuko Nakamura; Eri Suzuki; Kazuhi πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 802 KB

## Abstract Microglia are the primary immune surveillance cells in the brain, and when activated they play critical roles in inflammatory reactions and tissue repair in the damaged brain. Microglia rapidly extend their processes toward the damaged areas in response to stimulation of the metabotropi

CD95-induced JNK activation signals are
✍ Thomas G. Hofmann; Andreas MΓΆller; Steffen P. Hehner; Dominik Welsch; Wulf DrΓΆge πŸ“‚ Article πŸ“… 2001 πŸ› John Wiley and Sons 🌐 French βš– 161 KB

Here we investigated CD95-mediated JNK activation pathways and their physiological relevance by employing a variety of cell lines with deficiencies in individual signal transmitting proteins. JNK activation was completely dependent on the activation of caspases in type I and type II cells, as reveal