𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Mechanisms of relapse in acute leukaemia: involvement of p53 mutated subclones in disease progression in acute lymphoblastic leukaemia

✍ Scribed by Zhu, Y-M; Foroni, L; McQuaker, I G; Papaioannou, M; Haynes, A; Russell, H H


Book ID
110001045
Publisher
Nature Publishing Group
Year
1999
Tongue
English
Weight
324 KB
Volume
79
Category
Article
ISSN
0007-0920

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Hepatotoxicity in the treatment of acute
✍ Topley, J. M. ;Benson, J. ;Squier, M. V. ;Chessells, J. M. πŸ“‚ Article πŸ“… 1979 πŸ› John Wiley and Sons 🌐 English βš– 368 KB

Serial liver function tests and percutaneous liver biopsies were performed on 21 children receiving treatment for acute lymphoblastic leukaemia (ALL). The patients received continuing chemotherapy either with daily 6-mercaptopurine and weekly methotrexate or with five-day pulses of these drugs every

Mutational analysis of Bax and Bcl-2 in
✍ Gajja S. Salomons; Corine K. M. Buitenhuis; Cristina MartΓ­nez MuΓ±oz; Manon Verwi πŸ“‚ Article πŸ“… 1998 πŸ› John Wiley and Sons 🌐 French βš– 115 KB

In childhood acute lymphoblastic leukaemia there are large interpatient variations in levels of the apoptosis-regulating proteins Bax and Bcl-2, but the molecular basis for this variation is unknown. Point-mutations in bax have been reported in cell lines derived from haematological malignancies. Fr