T-cell-dependent B-cell help is likely to be of major importance in the pathogenesis of myasthenia gravis, but mechanisms provoking a pathological anti-acetylcholine receptor (AChR) response are poorly understood. We report on the dysregulation of recently identified CD4 Ψ /CD8 Ψ (double-negative) T
β¦ LIBER β¦
Mature, long-lived CD4+ and CD8+ T cells are generated by the thymoma in myasthenia gravis
β Scribed by Camilla Buckley; Daniel Douek; John Newsom-Davis; Angela Vincent; Nicholas Willcox
- Publisher
- John Wiley and Sons
- Year
- 2001
- Tongue
- English
- Weight
- 231 KB
- Volume
- 50
- Category
- Article
- ISSN
- 0364-5134
- DOI
- 10.1002/ana.1017
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