Maternal stress affects postnatal growth and the pituitary expression of prolactin in mouse offspring
- Publisher
- John Wiley and Sons
- Year
- 2011
- Tongue
- English
- Weight
- 46 KB
- Volume
- 89
- Category
- Article
- ISSN
- 0360-4012
No coin nor oath required. For personal study only.
β¦ Synopsis
The authors have submitted revisions for text on the following pages. Corrected sentences listed below:
Pages 329-330, sentence revised as:
In particular, we focused on the expression of prolactin (PRL), because changes in PRL expression in the maternal hypothalamus were previously reported to induce persistent detrimental effects on body weight control of the pups (Bonomo et al., 2007(Bonomo et al., , 2008)).
Sentence revised in the section: Gene Expression Analysis Primer sets (IGF-1, GH, OXT, PRL, LHb and GAPDH) were designed (Table I) for use in TaqMan gene expression assays (Applied Biosystems, Foster City, CA) or Custom Taq-Man gene expression assays (Applied Biosystems). The primer sets for the other genes (TSHb, aGSU, ACTH, FSHb, PIT1, PITX2a/b, PITX2c, and PROP1) were retrieved from published papers.
π SIMILAR VOLUMES
## Abstract Insulinβlike growth factorβ1 (IGFβ1) is essential to hippocampal neurogenesis and the neuronal response to hypoxia/ischemia injury. IGF (IGFβ1 and β2) signaling is mediated primarily by the type 1 IGF receptor (IGFβ1R) and modulated by six highβaffinity binding proteins (IGFBP) and the