𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Mapping of the gene encoding bovine preproacrosin (ACR) to Chromosome BTA 5 region q35

✍ Scribed by R. Friedl; I. M. Adham; O. J. Rottmann

Publisher
Springer-Verlag
Year
1994
Tongue
English
Weight
394 KB
Volume
5
Category
Article
ISSN
0938-8990

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

The gene encoding the human preproacrosi
The gene encoding the human preproacrosin (ACR) maps to the q13-qter region on chromosome 22
✍ I. M. Adham; K. -H. Grzeschik; A. H. M. Geurts van Kessel; W. Engel πŸ“‚ Article πŸ“… 1989 πŸ› Springer 🌐 English βš– 346 KB

Human proproacrosin is specified by a single gene (ACR). Using a series of human-rodent somatic cell hybrids containing variant complements of human chromosomes, the preproacrosin gene was found to cosegregate with human chromosome 22. Somatic cell hybrids containing translocation chromosomes carryi

Assignment of the human 8.5 H gene to ch
Assignment of the human 8.5 H gene to chromosome 5, region 5q35
✍ S. Diriong; D. SabΓ©ran-Djoneidi; M. LΓ©vi-Strauss; A. Bernheim; V. C. Nguyen πŸ“‚ Article πŸ“… 1994 πŸ› Springer 🌐 English βš– 442 KB

The human 8.5 H probe was isolated from a human cerebellum cDNA library with a probe corresponding to the coding region of the murine 8.5 M cDNA. This cDNA isolated from a routine cDNA library constructed from newborn cerebral hemispheres was selected because of its strong expression in embryonic ne

The human skeletal muscle adenine nucleo
The human skeletal muscle adenine nucleotide translocator gene maps to chromosome 4q35 in the region of the facioscapulohumeral muscular dystrophy locus
✍ Cisca Wijmenga; Sara T. Winokur; George W. Padberg; Mette I. Skraastad; Michael πŸ“‚ Article πŸ“… 1993 πŸ› Springer 🌐 English βš– 726 KB

Facioscapulohumeral muscular dystrophy (FSHD) is a relatively common autosomal dominant neuromuscular disorder. The gene for FSHD has recently been assigned to chromosome 4q35. Although abnormal mitochondrial and biochemical changes have been observed in FSHD, the molecular defect is unknown. In add