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M1959 Helicobacter pylori cagA Tyrosine Phosphorylation Triggers the Interleukin-11/STAT3 Pathway in Gastric Epithelial Cells

✍ Scribed by Lee, Kai Syin; Kalantzis, Anastasia; Murata-Kamiya, Naoko; Hatakeyama, Masanori; Giraud, Andrew S.; Menheniott, Trevelyan

Book ID: 123470928
Publisher: Elsevier Science
Year: 2010
Tongue: English
Weight: 50 KB
Volume: 138
Category: Article
ISSN: 0016-5085
DOI: 10.1016/S0016-5085(10)62069-0

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## Abstract Infection with CagA‐positive __Helicobacter pylori__ is the strongest risk factor for gastric carcinoma. Upon delivery into gastric epithelial cells, CagA disturbs cellular functions by physically interacting with and deregulating intracellular signaling molecules via both tyrosine phos