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Long-term cold ischemia reduces nitric oxide metabolism in reperfused rabbit kidneys

✍ Scribed by T.N. Hansen; A. D'Alessandro; J.H. Southard


Book ID
117235363
Publisher
Elsevier Science
Year
1997
Tongue
English
Weight
276 KB
Volume
29
Category
Article
ISSN
0041-1345

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## Abstract Nitric oxide (NO), originally identified as the mediator of endothelial‐dependent relaxation of vascular smooth muscle, is now known to also have cytotoxic effects under certain conditions. Thus, we have investigated the effects of inhibition of NO synthesis on ischemia/reperfusion inju