✦ LIBER ✦

Lipids and oxidised lipids in human atherosclerotic lesions at different stages of development

✍ Scribed by Keri L.H. Carpenter; Susan E. Taylor; Carina van der Veen; Bridget K. Williamson; James A. Ballantine; Malcolm J. Mitchinson

Book ID: 115741309
Publisher: Elsevier Science
Year: 1995
Tongue: English
Weight: 986 KB
Volume: 1256
Category: Article
ISSN: 0005-2760
DOI: 10.1016/0005-2760(94)00247-v

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Presence of oxidatively modified lipids

Presence of oxidatively modified lipids in human atherosclerotic lesions

✍ S. Bradamante; L. Barenghi; G. Giudici; G. Capaldi; L. Gabrielli; C. Vergani 📂 Article 📅 1990 🏛 Elsevier Science 🌐 English ⚖ 120 KB

Micellar organization of lipid droplets

Micellar organization of lipid droplets in human atherosclerotic lesions

✍ D. Kaul; V. Kothekar; R.K. Mishra 📂 Article 📅 1980 🏛 Elsevier Science 🌐 English ⚖ 287 KB

Incorporation of different fatty acids i

Incorporation of different fatty acids into combined lipids in rabbit atherosclerotic lesions

✍ A.J. Day; M.L. Wahlqvist; R.K. Tume 📂 Article 📅 1970 🏛 Elsevier Science 🌐 English ⚖ 932 KB

Lipid peroxidation and vitamin E in huma

Lipid peroxidation and vitamin E in human coronary atherosclerotic lesions

✍ Wojciech Jacheć; Andrzej Tomasik; Wojciech Ceglarek; Stanisław Woś; Jan Wodnieck 📂 Article 📅 2003 🏛 Elsevier Science 🌐 English ⚖ 149 KB

Phospholipids and oxophospholipids in at

Phospholipids and oxophospholipids in atherosclerotic plaques at different stages of plaque development

✍ Amir Ravandi; Saeid Babaei; Ramsey Leung; Juan Carlos Monge; George Hoppe; Henry 📂 Article 📅 2004 🏛 Springer-Verlag 🌐 English ⚖ 262 KB

Immunochemically detectable lipid-free a

Immunochemically detectable lipid-free apo(a) in plasma and in human atherosclerotic lesions

✍ Henry F. Hoff; June O'Neil; Gary B. Smejkal; Akira Yashiro 📂 Article 📅 1994 🏛 Elsevier Science 🌐 English ⚖ 741 KB

Although Lp(a) is an independent risk factor for cardiovascular diseases in humans, the precise pathogenetic mechanisms are still unknown. We have shown that Lp(a) accumulates in human atherosclerotic lesions, and some particles undergo oxidation. Since, following agarose electrophoresis of both pla