In six infants aged between 5 and 8 months with vitamin D deficient rickets, we have studied blood levels of calcium (Ca), phosphorus (P), alkaline phosphatase, immunoreactive parathyroid hormone (PTH) and calcitonin (CT), as well as urinary excretion of Ca, P, hydroxyproline and cyclic AMP, both un
Lactase activity in experimental rickets and vitamin D intoxication
β Scribed by Berio, A. ;Vogel, A. ;Lamedica, G. M. ;Nordio, S.
- Publisher
- Springer-Verlag
- Year
- 1968
- Weight
- 156 KB
- Volume
- 148
- Category
- Article
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract Children with calciumβdeficiency rickets may have increased vitamin D requirements and respond differently to vitamin D~2~ and vitamin D~3~. Our objective was to compare the metabolism of vitamins D~2~ and D~3~ in rachitic and control children. We administered an oral single dose of vit
Hereditary vitamin D-resistant rickets (HVDRR) is a rare autosomal recessive disease caused by mutations in the vitamin D receptor (VDR). Patients exhibit severe rickets and hypocalcemia. Heterozygous parents and siblings appear normal and exhibit no symptoms of the disease. We analyzed the VDR gene