Ca2+ release from skeletal sarcoplasmic reticulum (SR) could be regulated by at least three mechanisms: 1) Ca2+, 2) calmodulin, and 3) Ca2+/calmodulin-dependent phosphorylation. Bell-shaped Ca(2+)-dependence of Ca2+ release from both actively- and passively-loaded SR vesicles suggest that opening an
โฆ LIBER โฆ
Isolation of a Ca2+-releasing factor from caffeine-treated skeletal muscle fibres and its effect on Ca2+release from sarcoplasmic reticulum
โ Scribed by Annegret Herrmann-Frank; Gerhard Meissner
- Publisher
- Springer Netherlands
- Year
- 1989
- Tongue
- English
- Weight
- 912 KB
- Volume
- 10
- Category
- Article
- ISSN
- 0142-4319
No coin nor oath required. For personal study only.
๐ SIMILAR VOLUMES
Regulation of Ca2+release from sarcoplas
โ
Do Han Kim; Young Sup Lee; Arthur B. Landry
๐
Article
๐
1992
๐
Springer
๐
English
โ 321 KB
Effects of intracellular Ca2+chelating c
โ
P. Lipp; L. Pott
๐
Article
๐
1991
๐
Springer
๐
English
โ 964 KB
Ca2+ release from the sarcoplasmic reticulum (SR) of mammalian cardiac myocytes occurring either due to activation by a depolarization or the resulting transmembrane Ca2+ current (ICa), or spontaneously due to Ca2+ overload has been shown to cause inward current(s) at negative membrane potentials. I