𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Involvement of filamentous actin in setting the threshold for degranulation in mast cells

✍ Scribed by Helena Tolarová; Lubica Dráberová; Petr Heneberg; Petr Dráber


Publisher
John Wiley and Sons
Year
2004
Tongue
English
Weight
322 KB
Volume
34
Category
Article
ISSN
0014-2980

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Caldesmon is necessary for maintaining t
✍ Deng, Maoxian ;Mohanan, Sunish ;Polyak, Erzsebet ;Chacko, Samuel 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 830 KB

## Abstract Caldesmon (CaD), a component of microfilaments in all cells and thin filaments in smooth muscle cells, is known to bind to actin, tropomyosin, calmodulin, and myosin and to inhibit actin‐activated ATP hydrolysis by smooth muscle myosin. Thus, it is believed to regulate smooth muscle con

Mast cells migrate, but do not degranula
✍ Elektra J. Papadopoulos; David J. Fitzhugh; Christine Tkaczyk; Alasdair M. Gilfi 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 English ⚖ 338 KB 👁 1 views

Mast cells (MC) are anatomically located near nerves and blood vessels in skin and the gastrointestinal tract and tend to localize within certain cutaneous tumors such as neurofibromas. However, the molecular mechanisms by which MC home to these sites are not well characterized. Fractalkine (FK) is

Evidence of a threshold effect for TSP i
✍ Mark J. Nicolich; John F. Gamble 📂 Article 📅 1999 🏛 John Wiley and Sons ⚖ 237 KB

The purpose of the present paper is to examine the complex set of questions air pollution about the PM-mortality exposure-response (E-R) relationship by looking at the daily mortality practical and demonstrable consequences of time series model building and by exposure-response considering alternate

Comparison of FcϵRI- and FcγRI-mediated
✍ Yoshimichi Okayama; Christine Tkaczyk; Dean D. Metcalfe; Alasdair M. Gilfillan 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 318 KB

## Abstract We have demonstrated that CD34^+^ IFN‐γ‐treated human mast cells (HuMC) express functional FcγRI and that aggregation of these receptors leads to mediator release. As the signaling pathways linking FcγRI aggregation to mediator release are unknown, we examined FcγRI‐dependent activation