𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Investigation of the mechanisms of tissue factor-mediated evasion of tumour cells from cellular cytotoxicity

✍ Scribed by Chao Li; Mary E. W. Collier; G. Alkisitis Frentzou; John Greenman; Camille Ettelaie

Book ID
105967391
Publisher
Springer-Verlag
Year
2008
Tongue
English
Weight
377 KB
Volume
57
Category
Article
ISSN
0340-7004

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Mechanisms of cellular cytotoxicity medi
Mechanisms of cellular cytotoxicity mediated by effector cells from rats with spontaneous tumors
✍ M. ZΓΆller; S. Matzku πŸ“‚ Article πŸ“… 1982 πŸ› John Wiley and Sons 🌐 French βš– 516 KB

## Abstract We have previously shown that effector cells from BD X rats bearing spontaneous tumors display increased cytotoxicity towards syngeneic tumor cells compared to effector cells from untreated rats (ZΓΆller and Matzku, 1980__a__). The increased __in vitro__ cytotoxicity of lymphoid cells fr

Mast cells and tumour angiogenesis: The
Mast cells and tumour angiogenesis: The tumour-mediated release of an endothelial growth factor from mast cells
✍ William R. Roche πŸ“‚ Article πŸ“… 1985 πŸ› John Wiley and Sons 🌐 French βš– 780 KB

Exposure to tumour cells has previously been shown to induce mast cells to degranulate and release heparin. Isolated mastsell granules were found to be mitogenic for endothelial cells in vitro. This effect was a property of mast-cell heparin, whose potency as a mitogen exceeded that of commercial he

Selective induction of endothelial cell
Selective induction of endothelial cell tissue factor in the presence of a tumour-derived mediator: A potential mechanism of flavone acetic acid action in tumour vasculature
✍ J. Clifford Murray; Matthias Clauss; Juliana Denekamp; David Stern πŸ“‚ Article πŸ“… 1991 πŸ› John Wiley and Sons 🌐 French βš– 747 KB

Flavone acetic acid (FAA) is a potentially useful antitumour agent which has been reported to induce changes in tumour vasculature, in particular loss of bloodflow. This led us to examine whether endothelium could be a cellular target of FAA action, with resultant modulation of cell-surface coagulan