Microglial interaction with β-amyloid: I
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Maria E. Bamberger; Gary E. Landreth
📂
Article
📅
2001
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John Wiley and Sons
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English
⚖ 162 KB
## Abstract The etiology of Alzheimer's disease (AD) involves a significant inflammatory component as evidenced by the presence of elevated levels of a diverse range of proinflammatory molecules in the AD brain. These inflammatory molecules are produced principally by activated microglia, which are