✦ LIBER ✦

Intranigral Dopamine Toxicity andα-Synuclein Response in Rats

✍ Scribed by Cristina Gómez-Santos; Pol Giménez-Xavier; Isidre Ferrer; Santiago Ambrosio

Book ID: 106483449
Publisher: Springer
Year: 2006
Tongue: English
Weight: 294 KB
Volume: 31
Category: Article
ISSN: 0364-3190
DOI: 10.1007/s11064-006-9090-2

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

α-Synuclein overexpression increases dop

α-Synuclein overexpression increases dopamine toxicity in BE(2)-M17 cells

✍ Marco Bisaglia; Elisa Greggio; Dragan Maric; David W Miller; Mark R Cookson; Lui 📂 Article 📅 2010 🏛 BioMed Central 🌐 English ⚖ 368 KB

Aging enhances the neuroinflammatory res

Aging enhances the neuroinflammatory response and α-synuclein nitration in rats

✍ Dong-Young Choi; Jinlu Zhang; Guoying Bing 📂 Article 📅 2010 🏛 Elsevier Science 🌐 English ⚖ 459 KB

Dopamine and Cu +/2+ can induce oligomer

Dopamine and Cu +/2+ can induce oligomerization of α-synuclein in the absence of oxygen: Two types of oligomerization mechanisms for α-synuclein and related cell toxicity studies

✍ Ha, Yonghwang; Yang, Aerin; Lee, Seyoung; Kim, Kibong; Liew, Hyunjeong; Lee, San 📂 Article 📅 2013 🏛 John Wiley and Sons 🌐 English ⚖ 520 KB

Impairment of microtubule system increas

Impairment of microtubule system increases α-synuclein aggregation and toxicity

✍ Myunghye Kim; Wonsuk Jung; In-Hwan Lee; Ghibom Bhak; Seung R. Paik; Ji-Sook Hahn 📂 Article 📅 2008 🏛 Elsevier Science 🌐 English ⚖ 730 KB

Geldanamycin induces Hsp70 and prevents

Geldanamycin induces Hsp70 and prevents α-synuclein aggregation and toxicity in vitro

✍ Pamela J. McLean; Jochen Klucken; Youngah Shin; Bradley T. Hyman 📂 Article 📅 2004 🏛 Elsevier Science 🌐 English ⚖ 449 KB

RNA interference-mediated knockdown of α

RNA interference-mediated knockdown of α-synuclein protects human dopaminergic neuroblastoma cells from MPP+ toxicity and reduces dopamine transport

✍ Timothy M. Fountaine; Richard Wade-Martins 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 634 KB

## Abstract The critical observation in the pathology of Parkinson's disease (PD) is that neurodegeneration is largely restricted to dopaminergic neurons that develop cytoplasmic inclusions called Lewy bodies. These aggregations contain the protein α‐synuclein. Furthermore, it is becoming apparent