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Interleukin-4 inhibits RANKL-induced expression of NFATc1 and c-Fos: A possible mechanism for downregulation of osteoclastogenesis

✍ Scribed by Saad Gad Kamel Mohamed; Eiji Sugiyama; Kouichiro Shinoda; Hiroyuki Hounoki; Hirofumi Taki; Muneharu Maruyama; Tatsuro Miyahara; Masashi Kobayashi

Book ID
116290073
Publisher
Elsevier Science
Year
2005
Tongue
English
Weight
615 KB
Volume
329
Category
Article
ISSN
0006-291X

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Interleukin-4 inhibits RANKL-induced NFA
Interleukin-4 inhibits RANKL-induced NFATc1 expression via STAT6: A novel mechanism mediating its blockade of osteoclastogenesis
✍ Jing Cheng; Jianzhong Liu; Zhenqi Shi; Duorong Xu; Shaokai Luo; Gene P. Siegal; 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 532 KB

## Abstract Interleukin‐4 (IL‐4) is an important immune regulatory protein that possesses potent anti‐osteoclastogenic properties, and does so via the transcription factor STAT6. Previous studies have shown that IL‐4 selectively blocks RANKL‐induced activation of NF‐κB and mitogen‐activated protein