## Abstract The activation of the NF‐κB pathway by pro‐inflammatory cytokines, such as tumor necrosis factor‐α (TNFα), can be an important contributor for the re‐programming of chondrocyte gene expression, thereby making it a therapeutic target in articular diseases. To search for new approaches to
Interleukin-1β and tumor necrosis factor α inhibit chondrogenesis by human mesenchymal stem cells through NF-κB–dependent pathways
✍ Scribed by N. Wehling; G. D. Palmer; C. Pilapil; F. Liu; J. W. Wells; P. E. Müller; C. H. Evans; R. M. Porter
- Publisher
- John Wiley and Sons
- Year
- 2009
- Tongue
- English
- Weight
- 956 KB
- Volume
- 60
- Category
- Article
- ISSN
- 0004-3591
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