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Interference of dexamethasone with leukocyte blood volume and albumin movements in lungs from endotoxemic guinea-pigs

✍ Scribed by C.M.M.B. de Castro; M.F. Bureau; B.B. Vargaftig; M. Bachelet


Publisher
Elsevier
Year
1995
Tongue
English
Weight
777 KB
Volume
8
Category
Article
ISSN
0952-0600

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✦ Synopsis


Using radioactive tracers, we measured blood volume, albumin exchanges and blood leukocyte sequestration within lungs, following an intravenous injection of lipopolysaccharide (0.1-1 mg/kg). Neutrophil infiltration into the airways was followed in parallel experiments. Dexamethasone pretreatment (20 mg/kg, subcutaneous) failed to prevent early pulmonary changes induced by lipopolysaccharide as decreased blood volume, leukocyte sequestration, leukopenia or the increased trans-endothelial albumin exchanges. However, dexamethasone provided a significant protection against the later albumin leakage through the endothelial/epithelial barrier and the neutrophil accumulation in the airways observed in lipopolysaccharide-treated guinea-pigs. Our results indicate that the protective effect of dexamethasone in lipopolysaccharide-induced lung injury might derive from an initial reduction of leukocyte adhesion and a later decrease in alveolo-capillary permeability.