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Inhibition of GSK-3β promotes survival and proliferation of megakaryocytic cells through a β-catenin-independent pathway

✍ Scribed by Mie Soda; Karl Willert; Kenneth Kaushansky; Amy E. Geddis

Book ID: 113528241
Publisher: Elsevier Science
Year: 2008
Tongue: English
Weight: 527 KB
Volume: 20
Category: Article
ISSN: 0898-6568
DOI: 10.1016/j.cellsig.2008.09.001

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## Abstract Glycogen synthase kinase‐3β (GSK‐3β) and adenomatous polyposis coli (APC) play an important role in the regulation of β‐catenin. Inhibition of or defects in their functions can lead to activation of β‐catenin. β‐catenin has been recently found to interact with and inhibit nuclear factor