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Inhibition of Aβ fibril formation and Aβ-induced cytotoxicity by senile plaque-associated proteins

✍ Scribed by Akira Monji; Ichiro Yoshida; Ken-ichiro Tashiro; Yoshihito Hayashi; Kazunori Matsuda; Nobutada Tashiro

Book ID
117478267
Publisher
Elsevier Science
Year
2000
Tongue
English
Weight
427 KB
Volume
278
Category
Article
ISSN
0304-3940

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Alzheimer's disease (AD) is a progressive neurodegenerative disorder. Pathology visible "post mortem" includes neurodegeneration and extracellular deposition of amyloids, both in neuritic plaques and diffuse deposits. [1] The major proteinaceous component of AD amyloids is the amyloid-b protein (Ab)