𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Induction of the interleukin 1 receptor antagonist protein by transforming growth factor-β

✍ Scribed by Martin Turner; David Chantry; Peter Katsikis; Ann Berge; Fionula M. Brennan; Marc Feldmann

Publisher
John Wiley and Sons
Year
1991
Tongue
English
Weight
506 KB
Volume
21
Category
Article
ISSN
0014-2980

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Induction of transforming growth factor
Induction of transforming growth factor β1 by insulin-like growth factor-1 in dermal fibroblasts
✍ Aziz Ghahary; Qiong Shen; You J. Shen; Paul G. Scott; Edward E. Tredget 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 200 KB 👁 1 views

Transforming growth factor b1 (TGF-b1) belongs to a family of multifunctional modulatory proteins involved in cell growth, differentiation, development, and wound healing. Although the biological activities of TGF-b1 have been extensively studied, its regulation remains obscure. Here we report the e

Regulation of transforming growth factor
Regulation of transforming growth factor-β production by interleukin-12
✍ Thomas Marth; Warren Strober; Robert A. Seder; Brian L. Kelsall 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 847 KB

## Regulation of transforming growth factor-p production by interleukin-= The induction of peripheral tolerance following oral antigen administration in several autoimmune disease and conventional animal models correlates with the production of transforming growth factor-fJ (TGF-p) and T helper ty

Interleukin-4, interleukin-10, and inter
Interleukin-4, interleukin-10, and interleukin-1-receptor antagonist but not transforming growth factor-β induce ramification and reduce adhesion molecule expression of rat microglial cells
✍ Florentina Wirjatijasa; Faramarz Dehghani; Roman A. Blaheta; Horst-Werner Korf; 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 329 KB

The activity of microglial cells is strictly controlled in order to maintain central nervous system (CNS) immune privilege. We hypothesized that several immunomodulatory factors present in the CNS parenchyma, i.e., the Th2-derived cytokines interleukin (IL)-4 and IL-10, interleukin-1-receptor-antago