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Increased Inducible Nitric Oxide Synthase Expression Contributes to Myocardial Dysfunction and Higher Mortality After Myocardial Infarction in Mice

✍ Scribed by Feng, Q.; Lu, X.; Jones, D. L.; Shen, J.; Arnold, J. M. O.


Book ID
111937511
Publisher
Lippincott Williams and Wilkins
Year
2001
Tongue
English
Weight
799 KB
Volume
104
Category
Article
ISSN
0009-7322

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The role of nitric oxide (NO) in liver injury and fibrosis is unclear. The purpose of this study was to determine whether inducible NO synthase deficiency (iNOS(-/-)) affects liver injury and fibrosis produced in mice by chronic carbon tetrachloride (CCl(4)) administration. Wild-type (WT) or iNOS(-/