## Abstract ## Objective Interleukinβ1 (ILβ1) acts via its receptors to induce gene expression that mediates protein synthesis involved in inflammation. Increased expression of ILβ1Ξ± and ILβ1Ξ² in muscle tissue from patients with polymyositis and dermatomyositis has been demonstrated. It is not kno
β¦ LIBER β¦
Idiopathic inflammatory myopathy of the antisynthetase (jo-1) type associated with noncaseating granulomas
β Scribed by Kevin G. Moder; Thomas A. Gaffey; Eric L. Matteson
- Publisher
- John Wiley and Sons
- Year
- 1993
- Tongue
- English
- Weight
- 465 KB
- Volume
- 36
- Category
- Article
- ISSN
- 0004-3591
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## Communicated by Claude Fe Β΄rec Mutations of the ryanodine receptor cause dominant and recessive forms of congenital myopathies with cores. Quantitative defects of RYR1 have been reported in families presenting with recessive forms of the disease and epigenic regulation has been recently proposed