Hypovolaemic shock with oedema due to increased capillary permeability

Exposure of tumor necrosis factor-α to l

Exposure of tumor necrosis factor-α to luminal membrane of bovine brain capillary endothelial cells cocultured with astrocytes induces a delayed increase of permeability and cytoplasmic stress fiber formation of actin

✍ M. A. Deli; L. Descamps; M.-P. Dehouck; R. Cecchelli; F. Joó; C. S. Ábrahám; G. 📂 Article 📅 1995 🏛 John Wiley and Sons 🌐 English ⚖ 1009 KB

## Abstract Tumor necrosis factor‐α (TNF‐α), a proinflammatory cytokine, has long been known to be involved in the pathogenesis of central nervous system infections and of certain neurodegenerative diseases. However, the possible role of the blood‐brain barrier (BBB), the active interface between t