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Hyperpigmentation and melanocytic hyperplasia in transgenic mice expressing the human T24 HA-ras gene regulated by a mouse tyrosinase promoter

✍ Scribed by Marianne Broome Powell; Paul Hyman; O. Dianna Bell; Allan Balmain; Ken Brown; Dave Alberts; G. Tim Bowden


Publisher
John Wiley and Sons
Year
1995
Tongue
English
Weight
817 KB
Volume
12
Category
Article
ISSN
0899-1987

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✦ Synopsis


Abstract

The tyrosinase promoter has been used to target expression of the mutated human T24 Ha‐ras oncogene in pigment‐producing cells of transgenic mice. Two independent founder mice carrying the transgene survived and showed the same distinct phenotype of muted coat color, deeply pigmented skin with multiple nevi, and twirling behavior. The offspring of one of these founders were developed into a line that stably expressed the same phenotype. Histopathological analysis of the tissues revealed hyperpigmentation and/or melanocytic hyperplasia in the skin, eyes, inner ear, and meningeal membranes in the brain. Reverse transcriptase‐polymerase chain reaction analysis revealed expression of the transgene in skin, brain, and spleen. We propose that these transgenic mice will be a model for studying the process of multistage melanoma carcinogenesis and a system for evaluating potential chemopreventive agents. © 1995 Wiley‐Liss Inc.