As the molecular basis of Duchenne Muscular Dystrophy (DMD) was being discovered, increasing focus was placed on the mechanisms of progressive failure of myoregeneration. In this study, we propose a pathogenesis model for DMD, where an autocrine growth factor release of TGF-β€1-from necrotic myofiber
β¦ LIBER β¦
Human molecular genetics and the elucidation of the primary biochemical defect in duchenne muscular dystrophy
β Scribed by Hoffman, Eric P.
- Publisher
- John Wiley and Sons
- Year
- 1989
- Tongue
- English
- Weight
- 534 KB
- Volume
- 14
- Category
- Article
- ISSN
- 0886-1544
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Defective growth in vitro of Duchenne mu
β
Mariarosa A.B. Melone; Gianfranco Peluso; Orsolina Petillo; Umberto Galderisi; R
π
Article
π
2000
π
John Wiley and Sons
π
English
β 234 KB
π 1 views
Molecular analysis of the Duchenne muscu
β
PatiΓ±o, Ana ;Narbona, Juan ;GarcΓa-Delgado, Marina
π
Article
π
1995
π
John Wiley and Sons
π
English
β 506 KB
Introduction: The expansion of clinical
β
Kiichi Arahata; Hideo Sugita
π
Article
π
1995
π
John Wiley and Sons
π
English
β 238 KB
Cells of extraembryonic mesodermal origi
β
Yayoi Kawamichi; Chang-Hao Cui; Masashi Toyoda; Hatsune Makino; Akane Horie; Yor
π
Article
π
2010
π
John Wiley and Sons
π
English
β 604 KB
## Abstract Duchenne muscular dystrophy is an Xβlinked recessive genetic disease characterized by severe skeletal muscular degeneration. The placenta is considered to be a promising candidate cell source for cellular therapeutics because it contains a large number of cells and heterogenous cell pop
Molecular-genetic study of Duchenne and
β
Sugino, Shigeto ;Fujishita, Satoshi ;Kamimura, Naohisa ;Matsumoto, Tadashi ;Wape
π
Article
π
1989
π
John Wiley and Sons
π
English
β 657 KB