## Abstract Exposure to ultraviolet radiation (UVR) is a known risk factor for cataract, but the molecular mechanisms involved have not been elucidated. We hypothesized that exposure to UVR would modulate the activation of nuclear factor kappa‐B (NF‐κB) within the human lens epithelium, since NF‐κB
HLA–B27 modulates nuclear factor κB activation in human monocytic cells exposed to lipopolysaccharide
✍ Scribed by Markus A. Penttinen; Carina I. Holmberg; Lea Sistonen; Kaisa Granfors
- Publisher
- John Wiley and Sons
- Year
- 2002
- Tongue
- English
- Weight
- 278 KB
- Volume
- 46
- Category
- Article
- ISSN
- 0004-3591
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Exposure of cells to ionizing radiation (IR) determines cellular lesions, such as DNA and membrane damage, which involve a coordinate network of signal transduction pathways responsible for resistance to or delay of apoptosis, depending on cell type and administered dose. Since, after I
## Abstract The cytoskeleton in eukaryotic cells is composed of two major filament systems, the microtubule system and the actin cytoskeleton. The microtubule system has recently emerged as an important regulator of NF‐κB function. However, the role that the actin microfilament system plays in cont
## Abstract The cytoskeleton in eukaryotic cells is composed of two major filament systems, the microtubule system and the actin cytoskeleton. The microtubule system has recently emerged as an important regulator of NF‐κB function. However, the role that the actin microfilament system plays in cont
## Abstract It has been clearly established that receptor activator of nuclear factor kappa B ligand (RANKL) is a key cytokine involved in the differentiation of osteoclastic precursors of the monocytic/macrophagic lineage. However, relatively little information is available on the ability of RANKL