B virus in 2 cases, hepatitis C virus in 5), but who had not developed The statistical analysis was performed by G. Leandro, M.D., biostatistician. cirrhosis at the end of a comparable follow-up time (mean 30.1 Supported in part by a grant from the ''CCC'' Association Against Cancer, Cittadella, m
Hepatic stellate cells: Partners in crime for liver metastases?
β Scribed by Ningling Kang; Gregory J. Gores; Vijay H. Shah
- Publisher
- John Wiley and Sons
- Year
- 2011
- Tongue
- English
- Weight
- 445 KB
- Volume
- 54
- Category
- Article
- ISSN
- 0270-9139
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
The pathogenic mechanisms of accelerated graft fibrosis in hepatitis C recurrence after liver transplantation (LT) are not well established. The aim of the study was to assess whether a greater activation of hepatic stellate cells (HSC), the major collagen-producing cells in the liver, can occur in
Recurrent hepatitis C after liver transplantation is a serious problem faced by liver transplant recipients. Activation of hepatic stellate cells is an early step in hepatic fibrogenesis. The aim of this study was to evaluate hepatic stellate cell activation, early after liver transplantation, as a
procollagen type I, fibronectin, and laminin in the DMN Interferon gamma (IFN-g) inhibits in vitro the activa-/ IFN-g group. Thus, this study indicates that IFN-g retion of hepatic stellate cells (HSC), the primary extracelduces extracellular matrix deposition in vivo by inhibilular matrix-producing
Fibronectins are multifunctional glycoproteins that are important components of the extracellular matrix in normal and fibrotic liver. Multiple fibronectin isoforms are generated from a single gene by alternative splicing of the primary transcript at the domains EIIIA, EIIIB, and V. The aim of this