Genetic polymorphisms in DNA repair genes as modulators of Hodgkin disease risk
β Scribed by Randa El-Zein; Claudia M. Monroy; Carol J. Etzel; Andrea C. Cortes; Yun Xing; Amanda L. Collier; Sara S. Strom
- Publisher
- John Wiley and Sons
- Year
- 2009
- Tongue
- English
- Weight
- 143 KB
- Volume
- 115
- Category
- Article
- ISSN
- 0008-543X
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract Rates of adenocarcinoma of the esophagus (EAC) and esophagoβgastric junction (EGJAC) have increased rapidly in recent decades. The primary risk factors, gastroβesophageal acid reflux and smoking, are potentially genotoxic through the generation of __N__βnitroso compounds. The DNA repair
## Abstract Genetic instability plays a crucial role in cancer development. The genetic stability of the cell as well as DNA methylation status could be modulated by folate levels. Several studies suggested associations between polymorphisms in folate genes and alterations in protein expression and
## Abstract Highβdose ionizing radiation exposure to the breast and rare autosomal dominant genes have been linked with increased breast cancer risk, but the role of lowβtoβmoderate doses from protracted radiation exposure in breast cancer risk and its potential modification by polymorphisms in DNA