Genetic contribution of the tumor necrosis factor region in guillain-barré syndrome
✍ Scribed by J. J. Ma; Dr. M. Nishimura; H. Mine; S. Kuroki; M. Nukina; M. Ohta; H. Saji; H. Obayashi; H. Kawakami; T. Saida; T. Uchiyama
- Publisher
- John Wiley and Sons
- Year
- 1998
- Tongue
- English
- Weight
- 416 KB
- Volume
- 44
- Category
- Article
- ISSN
- 0364-5134
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✦ Synopsis
insular cortex is a likely candidate to mediate stressinduced cardiovascular responses." Thus, as far as our study is concerned, we believe that insular activation may reflect nonspecific autonomic efferent responses during the unpleasant stimulation procedure rather than specific visceral afferent processing.
The strongest and apparently most noxious stimulus led to activation of the anterior cingulate cortex just above the corpus callosum. This cortical area was reported to be responsive to noxious stimuli and involved with processing of both somatic and visceral pain.2 1-23 Likewise, area 24 might be involved in response selection such as nocifensor reflex inhibitionz4 or in emotional coping with noxious stimuli.25
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Therapeutic plasma exchange (TPE) is a standard treatment in Guillain-Barre ´syndrome. TPE may require exogenous fluid for replacement of plasma and, depending on the equipment used, varying extracorporeal volumes. Potential adverse effects include allergic reaction, infection, and hypotension. From