𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Genetic and epigenetic changes of HPV16 in cervical cancer differentially regulate E6/E7 expression and associate with disease progression

✍ Scribed by Dipanjana Mazumder (Indra); Ratnesh Kumar Singh; Sraboni Mitra; Sankhadeep Dutta; Chandraditya Chakraborty; Partha Sarathi Basu; Ranajit Kumar Mondal; Susanta Roychoudhury; Chinmay Kumar Panda

Book ID
113963409
Publisher
Elsevier Science
Year
2011
Tongue
English
Weight
665 KB
Volume
123
Category
Article
ISSN
0090-8258

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Characterization of humoral immune respo
Characterization of humoral immune responses against p16, p53, HPV16 E6 and HPV16 E7 in patients with HPV-associated cancers
✍ Miriam Reuschenbach; Tim Waterboer; Keng-Ling Wallin; Jens Einenkel; Joakim Dill πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 French βš– 241 KB πŸ‘ 1 views

## Abstract The cellular tumor suppressor p16 is strongly overexpressed in cervical cancers and precancers. We have previously demonstrated that infiltrating T lymphocytes reactive against p16 can be found in cervical cancer patients. Here, we analyzed whether p16 induces humoral immune responses.

Uniform distribution of HPV 16 E6 and E7
Uniform distribution of HPV 16 E6 and E7 variants in patients with normal histology, cervical intra-epithelial neoplasia and cervical cancer
✍ Ingo Nindl; Knuth Rindfleisch; Beatrix Lotz; Achim Schneider; Matthias DΓΌrst πŸ“‚ Article πŸ“… 1999 πŸ› John Wiley and Sons 🌐 French βš– 160 KB πŸ‘ 2 views

High-risk human papillomaviruses (HPV), particularly HPV 16, are associated with invasive cervical cancer (ICC), and persistent high-risk HPV infection is considered to be a marker for progressive cervical intra-epithelial neoplasia (CIN). However, most high-risk, HPV-infected, pre-cancerous lesions