𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Gastrin releasing peptide GRP(14–27) in human breast cancer cells and in small cell lung cancer

✍ Scribed by A. J. Vangsted; E. V. Andersen; L. Nedergaard; J. Zeuthen

Publisher
Springer US
Year
1991
Tongue
English
Weight
1015 KB
Volume
19
Category
Article
ISSN
0167-6806

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

GRP receptors are present in non small c
GRP receptors are present in non small cell lung cancer cells
✍ Dr. T. W. Moody; F. Zia; R. Venugopal; M. Fagarasan; H. Oie; V. Hu 📂 Article 📅 1996 🏛 John Wiley and Sons 🌐 English ⚖ 871 KB

Previously, GRP receptors were characterized in small cell lung cancer cells and here non-small cell lung cancer (NSCLC) cells were investigated: (1251-Tyr4) bombesin (BN) or lZ51-GRP bound with high affinity to NCLH720 (lung carcinoid) and NCI-HI 299 (large cell carcinoma) cells. Binding was specif

Characterization, in some human breast c
Characterization, in some human breast cancer cell lines, of gastrin-releasing peptide-like receptors which are absent in normal breast epithelial cells
✍ Sylvie Giacchetti; Christiane Gauvillé; Patricia De Crémoux; Laurent Bertin; Phi 📂 Article 📅 1990 🏛 John Wiley and Sons 🌐 French ⚖ 736 KB

## Abstract The binding of ^125^I‐Tyr^4^ bombesin was investigated on plasma membranes of 8 human breast cancer cell lines and 2 long‐term cultures of normal human breast epithelial cells. Scatchard plots were compatible with high‐affinity, single‐site class of receptors in 3 cell lines (K~D~ of 0.

Aberrant methylation of TMS1 in small ce
Aberrant methylation of TMS1 in small cell, non small cell lung cancer and breast cancer
✍ Arvind Virmani; Asha Rathi; Kenji Sugio; Ubaradka G. Sathyanarayana; Shinichi To 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 French ⚖ 320 KB 👁 2 views

## Abstract __TMS1__ (target of methylation‐induced silencing) is a CpG island‐associated gene that functions in the regulation of apoptosis and encodes a caspase recruitment domain, a recently described motif found in apoptotic signaling molecules. Recent evidence suggests that silencing of genes