Glutamate, the principal retinal neurotransmitter, can also act as a toxin when present in excessive concentrations as may occur in pathologies such as retinal ischemia or more generally in cerebral neuronal degenerative disease. As glial cells play pivotal roles in transfer of blood-borne molecules
Gap junction–mediated loops of neuronal-glial interactions
✍ Scribed by Maira M. Fróes; Antônio C. Campos De Carvalho
- Publisher
- John Wiley and Sons
- Year
- 1998
- Tongue
- English
- Weight
- 276 KB
- Volume
- 24
- Category
- Article
- ISSN
- 0894-1491
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