Gap junctional intercellular communication (GJIC) has been proposed as a cellular mechanism for tumour suppression and there is experimental evidence in support of this. If aberrant GJIC contributes to the formation of human breast tumours, one might expect that the connexins (gap junction proteins)
Gap Junction Distribution and Connexin Expression in Human Breast
β Scribed by Paul Monaghan; Catherine Clarke; Nina P. Perusinghe; David W. Moss; Xiao-Yuan Chen; W.Howard Evans
- Book ID
- 115602280
- Publisher
- Elsevier Science
- Year
- 1996
- Tongue
- English
- Weight
- 497 KB
- Volume
- 223
- Category
- Article
- ISSN
- 0014-4827
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