The pathogenesis of idiopathic amyotrophic lateral sclerosis (ALS) remains unknown, but accumulating evidence suggests a neuroexcitotoxic mechanism may have some credence. Glutamate-induced hyperexcitability of cortical or spinal motoneurons may be expected to manifest itself as a reduced threshold
β¦ LIBER β¦
Further evidence for corticomotor hyperexcitability in amyotrophic lateral sclerosis
β Scribed by Daisuke Naka; Kerry R. Mills
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- English
- Weight
- 190 KB
- Volume
- 23
- Category
- Article
- ISSN
- 0148-639X
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