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Functional evidence for a role of combinedCDKN2A(p16-p14ARF)/CDKN2B(p15) gene inactivation in malignant gliomas

✍ Scribed by M. Simon; Gertraud Köster; Anil G. Menon; Johannes Schramm


Publisher
Springer-Verlag
Year
1999
Tongue
English
Weight
164 KB
Volume
98
Category
Article
ISSN
0001-6322

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The cyclin-dependent kinase inhibitor 2a (Cdkn2a) locus encodes two distinct tumor suppressors, p16INK4a and p19ARF, whose functions interrelate in the regulation of cell proliferation as key components of the retinoblastoma and p53 pathways, respectively. In many types of cancer, alterations of Cdk