𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Fructose 2,6-bisphosphate and insulin stimulation of glycolysis in 3T3-L1 adipocytes

✍ Scribed by Valeria Vasta; Paola Bruni; Fabio Vannini; Marta Farnararo

Publisher
Elsevier Science
Year
1989
Tongue
English
Weight
588 KB
Volume
21
Category
Article
ISSN
0020-711X

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Nigericin inhibits insulin-stimulated gl
Nigericin inhibits insulin-stimulated glucose transport in 3T3-L1 adipocytes
✍ Chih-Ying Chu; Ying-Shun Kao; Jim C. Fong 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 187 KB

## Abstract We used nigericin, a K^+^/H^+^ exchanger, to test whether glucose transport in 3T3‐L1 adipocytes was modulated by changes in intracellular pH. Our results showed that nigericin increased basal but decreased insulin‐stimulated glucose uptake in a time‐ and dose‐dependent manner. Whereas

Somatotropin antagonism of insulin-stimu
Somatotropin antagonism of insulin-stimulated glucose utilization in 3T3-L1 adipocytes
✍ Kevin C. Glenn; Kathleen S. Rose; Gwen G. Krivi 📂 Article 📅 1988 🏛 John Wiley and Sons 🌐 English ⚖ 895 KB

It is well established that somatotropin (GH) antagonizes insulin action in vivo and that supraphysiologic concentrations of GH frequently result in insulin resistance and glucose intolerance. However, the demonstration of an anti-insulin activity by GH in vitro has been difficult. This study, there

Tunicamycin-mediated depletion of insuli
Tunicamycin-mediated depletion of insulin receptors in 3T3-L1 adipocytes
✍ O. M. Rosen; G. H. Chia; C. Fung; C. S. Rubin 📂 Article 📅 1979 🏛 John Wiley and Sons 🌐 English ⚖ 459 KB

## Abstract Tunicamycin, an antibiotic that inhibits protein glycosylation, elicited a rapid depletion of insulin binding activity at the surface of 3T3‐L1 adipocytes. Disappearance of insulin receptors occurred more rapidly in the presence of tunicamycin than when protein synthesis was inhibited b

Suppression of Glutamine:Fructose-6-phos
Suppression of Glutamine:Fructose-6-phosphate amidotransferase-1 inhibits adipogenesis in 3T3-L1 adipocytes
✍ Tusty-Jiuan Hsieh; Tzu Lin; Pei-Chen Hsieh; Min-Chun Liao; Shyi-Jang Shin 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 437 KB

## Abstract O‐linked __N__‐acetylglucosamine (O‐GlcNAc) protein modification has been implicated in the regulation of signaling pathways, cell function, and gene expression. Glutamine:fructose‐6‐phosphate amidotransferase‐1 (GFAT‐1) is the rate‐limiting enzyme in the hexosamine biosynthetic pathway

Insulin-like and non-insulin-like seleni
Insulin-like and non-insulin-like selenium actions in 3T3-L1 adipocytes
✍ Emma Heart; Chin K. Sung 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 201 KB

## Abstract In insulin‐sensitive 3T3‐L1 adipocytes, selenium stimulates glucose transport and antilipolysis and these actions of selenium, like insulin actions, are sensitive to wortmanin, an inhibitor of phosphatidylinositol‐3‐kinase (PI3K). Selenium stimulates PI3K activity that is sustained up t

Palmitate and oleate induction of acylat
Palmitate and oleate induction of acylation stimulating protein resistance in 3T3-L1 adipocytes and preadipocytes
✍ Yu Wen; HongWei Wang; Robin MacLaren; Jing Wu; HuiLing Lu; Katherine Cianflone 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 205 KB

## Abstract Acylation stimulating protein (ASP) stimulates triglyceride synthesis and glucose transport via its receptor C5L2. The aims were (i) to evaluate ASP response under insulin‐resistant conditions and (ii) to identify mechanisms of ASP resistance using 3T3‐L1 adipocytes and preadipocytes. O