𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Frequent deletion of MTS1/p16 gene and correlation with clinicopathological parameters in endometrial carcinoma

✍ Scribed by Chunxiao Zhou; Jianheng Sun; Shixin Lu; Shunqian Jin; Hailing Liu; Xiugui Sheng


Publisher
Chinese Anti-Cancer Association
Year
1998
Tongue
English
Weight
326 KB
Volume
10
Category
Article
ISSN
1000-9604

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Frequency of mutation and deletion of th
✍ K Biden; J Young; R Buttenshaw; J Searle; G Cooksley; D B Xu; B Leggett πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 476 KB

The tumor suppressor gene CDKN2A (MTS1/p16), located on chromosome 9p21, is inactivated in a variety of tumors including melanomas and tumors of the biliary tract, pancreas, and stomach. The aim of the present study was to determine whether this gene is inactivated in hepatocellular carcinoma (HCC).

Germ-line mutations of the p16INK4(MTS1)
✍ P Chaubert; R Gayer; A Zimmermann; C Fontolliet; B Stamm; F Bosman; P Shaw πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 901 KB

The molecular mechanisms of hepatocarcinogenesis recent study on potential familial HCC in Alaskan natives are poorly understood. Only very recently has there excluded mutation of the p53 gene as the responsible genetic been a suggestion of familial hepatocellular carcinoma lesion. 1 This suggests t

Disregulation of p16MTS1/CDK4I protein a
✍ Pedro Jares; Alfons Nadal,; Pedro L. FernΓ‘ndez; Magda Pinyol; Lluis HernΓ‘ndez; M πŸ“‚ Article πŸ“… 1999 πŸ› John Wiley and Sons 🌐 French βš– 130 KB πŸ‘ 1 views

To determine the relationship between p16 MTS1/CDK4I expression, gene inactivation and 9p21 loss of heterozygosity (LOH) in the development of laryngeal carcinomas, we have examined p16 MTS1/CDK4I protein and mRNA expression in a series of 7 normal and 36 tumoral tissues, and the presence of gene al