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Fibrinolytic Mechanisms of tPA, prouPA, Mutant prouPA and Their Implications for Therapeutic Thrombolysis

✍ Scribed by Victor Gurewich

Book ID
120717370
Publisher
Springer US
Year
2013
Tongue
English
Weight
515 KB
Volume
4
Category
Article
ISSN
1869-408X

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## Abstract Achondroplasia (ACH), thanatophoric dysplasia (TD) types I and II, hypochondroplasia (HCH), and severe achondroplasia with developmental delay and acanthosis nigricans (SADDAN) are all due to activating mutations in the fibroblast growth factor receptor 3 (__FGFR3__) gene. We review the