๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Fetal loss rate after chorionic villus sampling and subsequent amniocentesis

โœ Scribed by Brandenburg, H. ;Jahoda, M. G. J. ;Pijpers, L. ;Reuss, A. ;Kleyer, W. J. ;Wladimiroff, J. W.


Publisher
John Wiley and Sons
Year
1990
Tongue
English
Weight
255 KB
Volume
35
Category
Article
ISSN
0148-7299

No coin nor oath required. For personal study only.

โœฆ Synopsis


Abstract

Here we review a group of 82 patients who underwent both chorionic villus sampling (CVS) and amniocentesis in the same pregnancy during the period May 1984โ€“May 1988. A fetal loss rate of 2.5% is documented. This is not essentially different from the sum of fetal loss rates following each of the procedures separately (CVS, 1.9%; amniocentesis, 1.0%) established during the same period.


๐Ÿ“œ SIMILAR VOLUMES


Estimating the spontaneous loss of Down
โœ Isabelle C. Bray; David E. Wright ๐Ÿ“‚ Article ๐Ÿ“… 1998 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 183 KB

Recent publications concerning the prevalence of Down syndrome at times of chorionic villus sampling (CVS), amniocentesis and livebirth were reviewed with particular interest in estimates of spontaneous loss of Down syndrome fetuses during pregnancy. Data from several studies were combined to provid

Comparison of cell cultures, chromosome
โœ K. Sundberg; C. Lundsteen; J. Philip ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 148 KB

548 cell cultures and karyotypes obtained by early amniocentesis with filtration technique (EAF) at a mean gestational age of 12Y weeks were compared with 555 obtained by transabdominal chorionic villus sampling (CVS) at a mean gestational age of 11 weeks. The number of abnormal karyotypes, culture

Prenatal diagnosis of the carbohydrate-d
โœ Joanne Charlwood; Peter Clayton; Geoffrey Keir; Nasi Mian; Elisabeth Young; Brya ๐Ÿ“‚ Article ๐Ÿ“… 1998 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 173 KB ๐Ÿ‘ 1 views

Two pregnancies at risk for the carbohydrate-deficient glycoprotein syndrome Type 1A (CDG1A, phosphomannomutase deficient) were monitored by enzyme and genetic linkage analyses. The index case in both families had a proven deficiency of phosphomannomutase (PMM). An unaffected fetus was predicted in