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FAS deficiency reduces apoptosis, spares axons and improves function after spinal cord injury

✍ Scribed by S. Casha; W.R. Yu; M.G. Fehlings

Book ID
118441215
Publisher
Elsevier Science
Year
2005
Tongue
English
Weight
733 KB
Volume
196
Category
Article
ISSN
0014-4886

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## Abstract Spinal cord injury (SCI) is followed by a secondary degenerative process that includes cell death. We have previously demonstrated that the chemokine CXCL10 is up‐regulated following SCI and plays a critical role in T‐lymphocyte recruitment to sites of injury and inhibition of angiogene